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Academic literature published using Medicilon selenoamino acid medium products

2023-07-18
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Medicilon provide complete sets of M9 selenomethionine (SeMET) medium for IPTG-induced E.coli expression system, production of selenomethionine-labeled protein and protein crystallography using multi-wavelength anomalous diffraction (MAD).

Academic literature published using Medicilon selenoamino acid medium products.jpg

Nature. 2008 Oct 9;455(7214):822-5.

Nat Microbiol. 2020 May;5(5):706-714.

Nucleic Acids Res. 2019 Jun 4;47(10):5420-5428.

Nucleic Acids Res. 2016 Jan 29;44(2):595-607.

Nucleic Acids Res. 2012 Feb;40(4):1868-78.

Nucleic Acids Res. 2010 Jun;38(10):3432-40.

Nat Struct Mol Biol. 2014 Aug;21(8):686-695.

Nat Struct Mol Biol. 2012 Dec;19(12):1316-23.

Nat Struct Mol Biol. 2009 Apr;16(4):421-9.

Nat Chem Biol. 2018 May;14(5):466-473.

Nat Commun. 2018 Jan 9;9(1):112.

Nat Commun. 2016 Jan 5;7:10175.

Nat Commun. 2016 Apr 22;7:11343.

Nat Commun. 2014 Jun 13;5:4068.

Sci Adv. 2016 Oct 21;2(10):e1601317.

Mol Syst Biol. 2016 Dec 16;12(12):893.

Proc Natl Acad Sci U S A. 2012 Nov 6;109(45):18459-64.

Elife. 2018 May 24;7:e27829.

mBio. 2021 Mar 16;12(2):e03287-20.

mBio. 2021 Feb 9;12(1):e02705-20.

mBio. 2015 Aug 11;6(4):e00806.

Acta Crystallogr D Struct Biol. 2020 Feb 1;76(Pt 2):166-175.

Protein Sci. 2020 Mar;29(3):711-722.

FEBS J. 2016 Jun;283(12):2206-18.

FEBS J. 2008 Oct;275(19):4824-35.

Environ Microbiol. 2014 Feb;16(2):359-81.

J Mol Biol. 2022 May 15;434(9):167559.

J Mol Biol. 2020 Feb 14;432(4):878-896.

J Mol Biol. 2015 Mar 27;427(6 Pt B):1316-1334.

J Mol Biol. 2015 Jun 19;427(12):2229-43.

J Mol Biol. 2011 Jul 15;410(3):411-23.

J Mol Biol. 2010 Jan 22;395(3):491-503.

Front Cell Infect Microbiol. 2018 Oct 5;8:352.

J Biol Chem. 2021 Oct;297(4):101193.

J Biol Chem. 2019 Sep 6;294(36):13233-13247.

J Biol Chem. 2018 Oct 19;293(42):16142-16159.

J Biol Chem. 2016 Aug 12;291(33):17283-92.

J Biol Chem. 2016 Jul 1;291(27):14120-14133.

J Biol Chem. 2015 Jan 2;290(1):601-11.

J Biol Chem. 2015 Oct 23;290(43):26249-58.

J Biol Chem. 2015 Oct 30;290(44):26856-65.

J Biol Chem. 2007 Dec 21;282(51):37215-24.

J Biol Chem. 2006 Dec 8;281(49):38061-70.

J Biol Chem. 2012 May 4;287(19):15242-50.

J Biol Chem. 2011 Apr 29;286(17):15344-51.

J Biol Chem. 2011 Jul 22;286(29):26093-106.

J Biol Chem. 2011 Jan 21;286(3):1966-75.

J Biol Chem. 2011 Sep 30;286(39):34023-35.

J Virol. 2015 Sep;89(17):8828-39.

Structure. 2019 May 7;27(5):785-793.e5.

Structure. 2010 May 12;18(5):638-48.

Structure. 2010 Sep 8;18(9):1083-93.

RNA. 2012 Nov;18(11):2020-8.

Sci Rep. 2015 Oct 22;5:15660.

Biochem J. 2018 Dec 19;475(24):3963-3978.

Biochem J. 2016 May 1;473(9):1141-52.

Proteins. 2015 Dec;83(12):2319-25.

Proteins. 2015 Aug;83(8):1547-54.

Proteins. 2015 Feb;83(2):383-8.

Proteins. 2012 Oct;80(10):2469-75.

Proteins. 2012 May;80(5):1495-9.

Proteins. 2011 Aug;79(8):2566-77.

Proteins. 2010 May 15;78(7):1631-40.

Adv Protein Chem Struct Biol. 2008;75:85-105.

J Bacteriol. 2016 Jul 28;198(16):2263-74.

J Bacteriol. 2015 Nov 9;198(3):463-76.

J Bacteriol. 2010 Jun;192(11):2746-56.

Molecules. 2022 Apr 20;27(9):2655.

PLoS One. 2017 Feb 3;12(2):e0171606.

PLoS One. 2014 Jul 10;9(7):e101787.

PLoS One. 2013;8(1):e52934.

PLoS One. 2012;7(3):e33943.

PLoS One. 2011 Mar 3;6(3):e16934.

PLoS One. 2011;6(8):e14827.

PLoS One. 2011;6(8):e22995.

PLoS One. 2009 Oct 20;4(10):e7165.

PLoS One. 2018 Dec 18;13(12):e0207563.

Chembiochem. 2022 Jan 5;23(1):e202100414.

Biochemistry. 2009 Nov 10;48(44):10542-8.

Biochemistry. 2013 Sep 17;52(37):6525-36.

Chembiochem. 2012 Nov 5;13(16):2408-15.

J Struct Biol. 2010 Mar;169(3):304-11.

Mol Biochem Parasitol. 2011 Oct;179(2):69-79.

BMC Struct Biol. 2011 May 26;11:27.

Acta Crystallogr F Struct Biol Commun. 2014 Oct;70(Pt 10):1318-23.

J Struct Funct Genomics. 2014 Mar;15(1):33-40.

Acta Crystallogr D Biol Crystallogr. 2013 Jun;69(Pt 6):1073-89.

Acta Crystallogr Sect F Struct Biol Cryst Commun. 2013 Sep;69(Pt 9):956-61.

Acta Crystallogr Sect F Struct Biol Cryst Commun. 2012 Jan 1;68(Pt 1):89-92.

Acta Crystallogr Sect F Struct Biol Cryst Commun. 2012 Nov 1;68(Pt 11):1294-9.

Acta Crystallogr Sect F Struct Biol Cryst Commun. 2011 Nov 1;67(Pt 11):1399-402.

Acta Crystallogr Sect F Struct Biol Cryst Commun. 2009 Mar 1;65(Pt 3):260-3.

Acta Crystallogr Sect F Struct Biol Cryst Commun. 2008 Jun 1;64(Pt 6):554-7.

Acta Crystallogr Sect F Struct Biol Cryst Commun. 2008 Feb 1;64(Pt 2):98-101.

bioRxiv. 2023 May 12;2023.04.19.537381.

bioRxiv. January 23, 2023.

bioRxiv. November 26, 2022.

Journal of Molecular Biology. 2008 May 29, 379 (2):331-342.

University of Saskatchewan. October 2008.

Adam Mickiewicz University. Poznań, 2015.

McMaster University. Oct-2013.

McMaster University. Oct-2011.

Queen’s University. September, 2010.

Columbia University. January 22, 2014.

Adam Mickiewicz University. 24-lip-2015.

University of Calgary. 2021-01-29.

Virginia Tech University. 2021-11-15.

Virginia Tech University. 2013-06-11.

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